Well, hello, ladies and gents Robert Sykes Kido Savage.com.
And today, I've got special guest, Stephen hercy back on the
podcast. This is the fourth time.
I've had him on the show, every single time.
I talk to him. I'm like, man.
This could be like a three-hour episode because he is just
incredibly knowledgeable about all things heart, health diet.
He's type 1 diabetics. He brings a very unique approach
to nutrition. That just adds a whole nother
level of context there. And I just, I really appreciate
who he is, what he's doing, what information is putting on the
road. He's got a new book, Coming out
on April 7th, and I'm going to be 100% flipping of that thing,
cover to cover and learn as much as I can.
Because his knowledge of, you know, the heart and heart
health. Like I said is just
unparalleled. So very, very smart guy.
I have no doubt. They will take something from
this conversation. So that further Ado, sit back,
relax. Enjoy the podcast with Steven
hussy. We are live.
Steven has a how are you men doing?
Well, how are you? I'm good.
So I was, I was perusing back in
our prior podcast episodes and you've been on the show three
other times besides this was just kind of crazy.
The first one. I think we mostly do Vin to your
four pillars of evolution Evolution.
I think we talked quite a bit about that and then the last two
we touched on I think fourth phase water and how blood It's
to the the body independent of the pump, you know, the heart as
you said, it's not really referred to as a pump.
We drove into that. And then I think, in the last
episode, that was about a month after your heart attacks.
We talked about that. That's more or less.
I think where I didn't listen to all them, that's kind of what
we, what we covered in those episodes, but you've got a new
book coming out, you've done a lot since that last conversation
that we had in February of 2021. So just kind of bring us up to
speed man, it for you, but it hasn't listened to any of those
episodes which I encourage everybody to do.
Can you? It's like a quick little intro
or bio into you. Yeah, for sure.
So, yeah, I'm Steven has see I'm a chiropractor and functional
medicine practitioner. You know, my health Journey was
kind of inspired or my work is inspired by my health Journey
because at a very young age, I was diagnosed with a lot of
different chronic conditions, you know, everything from from
chronic hives to allergies to irritable bowel syndrome and
eventually ended up with autoimmune type 1, diabetes.
Which heavily predisposes me to heart disease and so throughout
all of my schooling and training and then in my own independent
research. I've been really interested in
heart disease. And so so I kind of dug into
that a lot. I've got quite a lot of
information and I decided to put it all down in this book and
became especially relevant to me when I had a heart attack, a
little over a year ago, at the age of 34 and she's really
trying to dive in and see why this happened to me and what I
found was pretty shocked. Because you know, first I was
like, I have this information is like, why couldn't I prevent
this? But then my experience in the
hospital and the care that I was given.
I mean, it was it was amazing what they're able to do to save
my life when that happened. But after the fact based on
everything, I knew I realized that their approach to Healing
my heart and preventing it from happening again.
Was was very pharmaceutical based and when you look at the
research, it's not very good based on that.
And so I wrote out that story in this book as Is like this big
introduction to why I'm so interested in heart disease and
all the kind of fascinating things that I found.
I summarized in this book and realized that, you know, based
on the care. I received in the hospital, this
it's information that I have. You know, first.
I was doubting myself, my God, can't, I can't release this book
or whatever, but then I realized that it's even more important.
When you release this information because in my
opinion, Western medicine is pretty lost.
When it comes to Healing the heart and and preventing these
types of things. Yeah.
I remember when you sent me. I guess it was like the
preliminary text to the book and you cause you had written a lot
of the book before the heart attack happened and the heart
attack happened. And you were kind of going
through this period of, you know, should I write this?
Should I put this out there? I mean, I feel kind of
hypocritical, etc. Etc.
So you wrote this really eloquent, you know, Memoir kind
of showcasing and illustrating all that and I feel like, you
know, going in hindsight now, the fact that you've had that
heart attack, it just puts your perspective on such another tear
that makes everything. You're saying that much more
relevant. Yeah, definitely.
And and you know, like I was faced with this decision, like
do I, you know, I just had a heart attack, which was, you
know, a big deal. Like it was a major heart attack
to and like, only twelve percent of people who have that type of
heart attack outside of the hospital survive.
And so, so I was, you know, very lucky to for those for the
people that did save my life, but, you know, going forward, I
was faced with this decision. Like do I follow the
recommendations? Which were basically take a
bunch of medications and and eat a eat a processed food.
It. Or do I?
Take? What all I've learned and
Implement those strategies and, and try and heal my heart in the
way that I've learned is best. And, and I'm happy to say that,
you know, that my, because this, I would have been able to talk
about this at the last time. I we had an interview because it
hadn't happened yet. But but yeah, I'm happy to say
that, my three-month echocardiogram.
My heart function is normal. It's back to normal.
Within like the ejection fraction of my heart is normal
and the Damage that was incurred into The middle of the septum of
my heart with a heart attack was was barely even present anymore.
Like it, it completely recovered and I did that only taking one
medication which was the blood thinner.
Now that I had a stint in an artery.
It was a good idea to be taking a blood thinner for a while
because we don't know how my body was going to react to that
stent. But other than that, I didn't
take any of the other medications and I implemented my
own health or my heart healing strategies.
And and the doctor was pretty amazed that in three months, my
heart was was he And functioning normally do you I mean was it
like a specific Catalyst that you could kind of pinpoint and
say this was likely the main contributing factor to causing
the heart attack or was there just a summation of multiple
different things? Yeah.
It was it was a summation of things.
I mean, type 1 diabetes heavily predisposes me and you know, I
can't say that I took care of myself in high school when I was
younger very well. I mean, I had very high when
sees, I didn't really understand the long-term repercussions in
that at that time, so that probably In help, but also, I
was I was under a lot of stress at the time and I got like, you
know, it was it was the year of covid and elections and all that
stuff. So all that stuff was getting at
me which are things. I usually wouldn't pay attention
to but since covid had this health related aspect to it.
I was I was kind of into it because health is kind of my
thing, but also I got some very stressful personal news.
The very beginning like two and a half days or one and a half
days before the heart attack happened.
Probably the most stressful days in my life.
And and so I think that there's no coincidence there.
But also I've been reading a little bit about a little bit
about exercise induced heart attacks.
And I and people who are already in an inflamed state.
So potentially I was with type 1 diabetes and all the stress.
I was undergoing at that time. I also you know the morning I
had the heart attack. I did a very intense workout,
which was my usual routine, like my kind of, like, hit training,
you know. Just going to complete
exhaustion in the exercises I was doing and I had a pretty
intense workout took a shower after that and then had a heart
attack and there are there is some evidence out there that in
an inflamed State. Like if someone's in an
unhealthy inflamed State and then they do that type of work
out that it can induce a clock because, you know, my CAC score
six months prior, which is the kind of amount of calcified
plaque in the arteries of the heart was Zero.
There was no calcified plaque in the artery of the heart, and And
the Interventional cardiologist that did my did my NGO and
place. The stent said that there was
pretty much no athletes grossest, in my coronary
arteries. It was just this giant cloth
that formed in the left anterior descending artery.
So if I was to suspect things, it was all that that stress and
build up the type 1 diabetes, that put me in this inflamed
State and then I did that work out.
And I induce this this clot and, which was super unfortunate, but
that's, that's the best that I can.
I can think of in the best that I could find looking into that.
I mean, you know, in the hospital they were all just
like, oh, your your cholesterol cause this or whatever and but
I, when I kept asking questions, they would just kind of dismiss
it and and wouldn't really open up the conversation to it.
But that's what I think. My best guess is what is what
happened? Were you just like was your skin
just boiling blood boiling skin crawling when you're in the
hospital and you're seeing what their standard of care is, and
what, you know, I mean knowing what, you know about the heart
and Your personal situation like were you on board with some of
the things they were doing? Are you totally opposed to most
of it? You know, I wouldn't say I was
totally opposed to, I mean, it's totally opposed to a lot of it
but not all of it. Like, I wanted their opinion,
you know, because I was just, I was just in this acute situation
and and I realized that one of the strengths of Western
medicine, is it an acute situation that can keep you
alive? And so I wanted to know what
they wanted to do after, you know, after they saved my Life.
And and but every time I would try and open up a discussion
with them about, well, what about the side effects of that?
Or is that the best thing for me?
They would just shut down the conversation and it's say,
either say I don't want to talk about it.
They'd say this is our recommendation and we're
sticking with it or they say, I don't know anything about that.
So I'm not going to talk about it because which is
understandable, because if they give me advice on something,
they don't know anything about it, you know, they're they're in
a hospital setting, they're all worried about lawsuits, you
know, right. And so I kind of respected some
of their answers, but it was still Discouraging to feel like
nobody wanted to listen, whether it was their own personal
reasoning, whether they want to listen, they just didn't care or
it was like other things preventing them from listening
because they were scared of lawsuits and that's how they've
been trained. But it was just a shutdown of
conversation. That was the most worrisome
thing to me wasn't necessary, cuz II know, Western medicine,
strengths and weaknesses, you know, I knew what to kind of
expect, you know, being in there, but it was just
frustrating that there was nobody really interested in what
I had to say. And and it was it was Mike are
you know, it wasn't like I was just some cookie cutter, heart
attack patient, you know, like they prescribed me a medication
that long term has been shown to cause insulin resistance of
square of type. 1 diabetics has diabetic is probably worse than
for a type to, you know, and so I was just like, why was that
prescribed rather than this other medication that did the
exact same thing that hasn't been shown to do that.
You know, like it was very clear.
They were treating my disease and not me as a whole person,
you know, and That was that was Troublesome.
But you know, that's where that's where I had to, you know,
rely on my knowledge to kind of say, okay.
I'm not going to do this. I am going to do this, but then
I realized that, you know, I'm a bit unique and then I've done
all this research for the heart disease and everything.
And so I know this stuff but there's people out there who are
going to be in the same situation.
I was in and they don't know this.
So I realized that, you know, yeah, I guess I really needed to
put the information out there because it may, it may be
life-saving for some other. People put them on a path that
will make it so that they don't end up back in that situation.
Because if I had followed some of the recommendations, I feel
like I'd be right back in and they were telling me to eat
completely processed food diet. And that was the best thing to
prevent heart. Failure, another heart attack
and I just knew that was absolutely, not true.
I mean, there was things like soy and fig newtons and and
Sherbert. And just all these processed
foods that I was allowed to eat on this diet and it was just it
was pretty disappointing. Yeah, I know.
Understand that, I mean, my wife Crystal and she was at the
hospital, you know, before, like when she drank it, all of her GI
stuff, figured out. They were recommending for
whatever reason. They just, like default to
really easily absorbed and digested foods, which always are
heavily processed high in carbohydrate sugary foods, but
it's like, why, why in the world of all the things out there is
that your primary recommendation is kind of what they tend to
default to. It seems Yeah, it's pretty
crazy. Like I was you know, they're
bringing me food while I was in the hospital.
I was there for three days but usually after a heart attack
like that, they keep people for a week, but I was I was doing a
lot better and I just asked to leave because I couldn't wait to
start eating real food again and start my own heart healing
routine. But yeah, they were bringing me
food and I'm trying to, like, avoid the most processed food.
So I'm just picking it things, but then it wasn't really
enough, you know, the things that were on my plate that I did
want to eat it. I would just end up being really
hungry. Sometimes I was just like, oh,
I'm just going to fast because it's probably better for my
healing anyways, and so so yeah, it was, it was a bit of a
struggle and it was during covid.
So no one was allowed to visit me.
I was alone the entire time as far as, you know, family or
friends being able to come in. So, one of my, one of my friends
did bring some stuff to the hospital and they brought it up
to me, but I wasn't allowed to see anybody.
So nobody could bring me food or or anything that I wanted.
And that was the other thing too is that I'm Type 1 diabetic and
and I have a very regimented way of controlling my blood sugar's
at my age. One.
Caesar, very good for a type 1 diabetic.
And despite, you know, seeing them, my agency was very good.
They insisted that they would that they should treat my type 1
diabetes, the way they wanted it to be done rather than what I
do. And they wouldn't let me control
demonstration of my insulin and things like that.
And so, my blood sugar's were very high.
And I kept saying, look, this is not working.
Let me do it. I know what to do and they kept
saying no, so when my friend brought me that that stuff and
they brought it up to me, there was insulin and then I started
managing myself and just said, no.
I'm not going to take yours. It's maddening.
It's crazy. And some of the nurses were even
like they were even like, I don't know why, they don't let
you type ones, just control things because it was under the
control of like the pharmacist in the end.
The doctor the attending, there is the, but some of the nurses
was just Like yeah, this happens all the time.
I don't know why, they don't let type ones, just control it
themselves, but it's because the hospital is liable.
Like, if I start administering my own drugs like that, they're
giving me then and if something goes wrong, then they're liable.
And that's just how it is. When in reality.
I was feeling terrible because my blood sugar was over 300 and
then at times because I just couldn't control it in the way
that they were trying to and so, and they wouldn't let me do it.
So but eventually like on the second day, I got that stuff and
I was able to get it down, control the way I wanted to, but
it Is maddening to me, you know, yeah, that's got to be
frustrating for sure. I mean, I totally get the
liability standpoint, you know, I mean, it makes sense what they
do and why they do it, but I wish there was an easy way to
bypass that like have you just simply sign a disclaimer or
waiver and then, you know let you do your own thing and if
they just Monitor and if it's obviously working then what
could they possibly have against it?
Yeah. Well, it's just crazy that that
in a place like a hospital where you think.
The number one goal is to get the patient's better bear.
Whatever means necessary. Terry.
There's all these there's all these things in place that are
protecting hospital and the doctors rather than what's best
for the patient. And that's just, I guess the
legal world we live in, but it's just interesting to see that
that that play out, you know. Yeah, it is crazy, man.
So ideally people will not have the perfect storm of cascading
negative events that lead to a heart attack like yours.
I've had several people on the podcast talk of ways, like
people can proactively monitor the health of their hearts and
their Arteries to hedge against that and some people are huge
advocates for the CAC scan. Some people are huge advocates
for look at the lipid markers and some people had dr.
Gundry on not too long ago and he was talking about a specific
lipid profile that measures inflammation.
I'm not sure what it was called again.
But is there anything that you personally recommend is like
just the best and most ensuring way to kind of get ahead of it
and proactively monitor things to make sure that you're not
likely going to have a heart attack.
Yeah, and so there's an interesting studies that I talk
about in my book that show. Well, they looked at, you know,
the traditional risk factors that people measure for heart
disease. So this is like an LDL or a
cholesterol panel or lipid panel or inflammatory markers, or high
blood pressure or various things like that, you know, and they
looked at those and they assess all the research that, that
looked at their those markers ability to predict whether Going
to have heart disease or a heart event or something like that.
And they found that they were very inadequate at predicting.
Those things in this paper. So that was pretty interesting
because I have my own set of things that I would want to look
at and it does include some of those, some of those risk
factors that are the more traditional type ones, you know,
but I look at them in the context of other things.
So if I take a lipid panel and look at that, I'd rather rather
than looking at levels. As of LDL, which I don't think
are very relevant when it comes to whether or not someone's
going to have heart disease or not.
Because I just I just posted a study into the other day on my
Instagram that showed that LDL level has no indication, whether
whether someone's going to develop atherosclerosis in the
arteries because they looked at a group of people and regardless
of LDL levels. Some of them had like it had low
LDL levels. Some of them still had
atherosclerosis and other ones, didn't, you know, so it didn't
really matter. So there's that.
But so when I Get a lipid panel. I look more like markers that
are going to tell me more about the person's metabolic health
because the person's metabolic health is going to indicate
whether or not their insulin resistant or not.
And so that's a big deal because there's always going to be
damaged a lot even artery. It's just normal wear and tear
of the body, you know, but whether or not someone gets
atherosclerosis when the the land the lining of the artery
gets damaged depends on that lining ability to repair itself,
and they Build it to repair itself, depends on insulin
signaling. And so if someone becomes
insulin-resistant, their metabolically unhealthy than
they, the the lining of the artery can't repair itself as
well. And we end up with the body of
doing something else which is depositing, cholesterol and
minerals that are trying to patch up this damage.
So it doesn't result in like a rupture of an artery or
something like that and that's atherosclerosis.
That's what that's the hardening of the arteries that we're
getting. And so looking for someone's
metabolic health and whether or not their insulin sensitive or
insulin, Sent it when more important.
So for that on a lipid panel, it's the trigged HDL ratio.
That's way more important and there's plenty of research
suggests that that's way more indicative of whether or not
someone's going to develop atherosclerosis or have a heart
event or something like that. But and then people talk about
all the different, all the different tests.
You can do, like looking at the particle size of the LDL and
looking at the small dense LDL or the lipoprotein, a or
oxidized LDL all these different Subfractions, I guess of these
lipids and those are all interesting but all those things
like it's not like the small dense LDL or causing after this
grossest. It's the things that cause small
dense LDL that also causes atherosclerosis and those are
things that damage tissues in the body.
So it's damaging LDL and creating small dense ldls are
oxidized ldls, but it's also damaging the lining of the
artery. So it's not that we want to like
the treatment should not be focused on reducing cholesterol.
Because they're small dense LDL, should be focused on reducing
the things that damage the cholesterol, which to me is
things that cause oxidative stress.
So oxidative stress is just when there's lots of free radicals in
the body that are that are causing damage to different
tissues in the body, including the lining of the artery.
And so we want to reduce those things.
And so for me, the first numbers you want to look at our
metabolic Health, but if you're if you have poor metabolic
Health that can lead oxidative stress, but then there's also
different things you can look at at when it comes to blood work
and things that are will measure your amount of oxidative stress
in your body, but they, you can get kind of in the weeds with
the easy. Another kind of obscure little
things that aren't normally taken, but you could take a GT,
which is a liver enzyme that kind of measures oxidative
stress on the liver. You can measure different things
like damage to different fatty acids or damage to DNA or levels
of glutathione or things like that.
That all can give you an idea if there's oxidative stress, but
Regardless of, if there's oxidative stress or not.
I'd still want to do the best, I can to reduce it and do things
to reduce it. Even if it's, I'm not testing
high for it, you know, because it's the oxidative stress and
inflammation that you could do. You could look at to to assess
whether or not there's going to be damage, the lining of
arteries, that may overwhelm the ability of the body to heal that
damaged. So you could also look at
markers of inflammation, like, you can look at various
cytokines, but, you know, the best General marker of
inflammation is like a heist. Sensitivity C-reactive protein
which is which is just definitely something that you
know, cardiologist do. Look at when you look at like a
cardiac pain all day. They measure that for sure
because that's definitely associated with with higher risk
of heart disease and things. But then, I think the thing that
is most underutilized as far as risk for heart disease is heart
rate, variability. That's just something that you
really don't find measure too much within Western medicine.
Is there a heart rate of It heart rate, variability that you
like to see kind of as a where's the line drawn between this is
good or this is bad in your mind.
Well, so there's really so anywhere from like 20 to 100,
is, is normal. So there's really no, like,
like, with heart rate variability.
There's really no, like, you don't want to sit there and
compare like someone yours or someone else's, you know, like
if yours is 80 in someone else's is 30, you know, those both are
normal. You really have to kind of track
it over time and find out what's normal.
For you. And what's what's your kind of
average is so that you can see when it changes, right?
Because when it's all kind of relative, I guess, because if I
if I sit at say 40 normally and then I'm tracking it and it
drops down to 20. It's like, okay, what happened?
That's not normal for me. You know, that's kind of how I
tell people to look at heart rate variability, because you
can't really just say, oh, I want to be in this range and In
the last chapter of my book. I kind of talked about how all
these different ranges, for different things.
On labs, are different biomarkers that we have may not
actually be as accurate as we think.
I mean, the ability of I Define health is the ability of the
body to adapt to certain situations.
And so trying to trying to quantify Health in a way that
puts everything in little boxes and it says, okay, it has to be
between these numbers is kind of kind of backwards when we look
at what what Fizz because health is like the ability of your body
to go out of those ranges and come back, right?
You know, that's that's what health is.
And so just like, you know, if you're if you like health
metabolically is if I eat a ton of carbohydrates and I take my
blood sugar out of you know, what could be considered
healthy, it does it come back easily and get back into
ketosis? After I do that.
That's metabolic Health, you know, and so just so if I ate a
bunch of carbohydrates and I My blood sugar and it was too high.
So she's like, oh, that's unhealthy.
Like well, no, that's what the body supposed to do move.
It comes back. That's how you know, or or
adaptation in the in autonomic nervous system.
Like, can I go through a stressful thing, a stressful
response and come back to Baseline, come back to balance
and the autonomic nervous system that's held, and that's what
heart rate variability is kind of measuring.
And so, if your if your heart rate variability is kind of in
this narrow window and it's lower than you don't have the
best ability to adapt to a stressful situation, which can
Too bad things happening in your body, you know, so so yeah, it's
interesting, you know to look at things like that because you
know blood Works never going to tell you if you're healthy.
I'm not saying that we should never take it or anything.
I'm just saying that you know, there's I know I've had plenty
of people who you know, come to me with blood work that is
completely off. So to speak.
Like things are way out of whack everything, but they feel
amazing. They have no symptoms
whatsoever. And then there's people who have
all kinds of symptoms and their blood looks Well, work looks
perfect, and they're just looking for explanations and
nobody can give it to him. So it's just this, it's this
tool. We use what we shouldn't, really
use it, or should we allow it to tell us a healthy?
Not we just use it as a part of an overall assessment on of our
health. But yeah, that's those are my
kind of things that look at someone's metabolically,
healthy. Look at the amount of oxidative
stress and inflammation and then definitely balancing the
autonomic nervous system by using heart rate, variability,
and make things better. You just using like a or ring or
something for HIV. Yeah.
I that's what I use to. Acts that, but there are lots of
different things. I mean, I don't know how
effective, a reliable, like the Apple watches are, but they'll
do it. But also, there's like a lead
HRV, there's different devices. People use like the Bayou straps
and just like something you can put on your wrist.
So there's lots of different devices that will help you track
that. Yeah.
I've got the word ring and I've been kind of keeping down HRV,
my wife Crystal. She's pregnant right now.
So her HRV is totally plummeted to like the teens, which is much
lower than normal, but I'm assuming that's probably normal
in the context of being. Pregnant, but mine's
consistently right around 50 or 60, which is pretty normal for
me. I'm probably sleep as much as I
should. But when it comes like that the
lipid profile, ma'am. I don't have any markers or
anything. That would be concerning.
Like, I look at mine. I'm consistently seeing Triggs
below, 50, H or HDL, you know, normally north of 100 and my
LDL, this is something that I'm curious about LDL seems to
fluctuate tremendously based. Of what people consume the night
before they get tested. So if they go in have like a,
you know, particularly fatty meal saturated fats, barbecue
for instance, and then do a 12-hour fast and go get their
Labs drawn the next day. Like that's typically going to
result in a significantly higher LDL count, which alarms their
their doctor, their General doc. So it just, it becomes a very
skewed glances to their health because it fluctuates
tremendously based off of something that, you know, they
had the night before. Yeah, definitely.
And and yeah, and you know, I'm sure you've heard of like day
Feldman and he's shown that, you know, if I if you if you don't
fast long enough then some of these numbers can be off.
So even a 12-hour fast may not be good enough.
If you're having, you know, High animal protein, high fat type of
meals, especially for triglycerides, but also for LDL,
so it's important not to say, you know, at least 14 hours for
some people in that situation. If you if you want to, you know,
get Truly accurate numbers because lots of times, you know,
those these more like keto meals and heavily more heavy animals.
They take a little longer to digest, which is not a problem.
But you have to account for that when you doing blood work, you
know, right. Right.
I think. I'm not sure what podcasts, I
listen to, I think it was saladino on Joe Rogan or
something. He was saying that, he's got LDL
north of 500. I want to say, is there a point
at which, like LDL exclusively being incredibly high as
alarming? Or is it all just It's part of
the bigger picture. I mean, I think the jury is kind
of out on that. I haven't seen anything to
suggest that having higher LDL like that high would be
problematic. But that doesn't mean that it
may be more of a result of nobody's really looked at it.
And I think that there are people.
Alright, right now, out there trying to design studies to look
at specifically that. But in my opinion, I don't see
how it would be, you know, if there was if there, if someone
was in a very inflamed Aid and had lots of oxidative stress and
having more LDL around, could be problematic, maybe it would,
maybe it would lead to, you know, less or higher viscosity
of the blood, which is more likely to induce clotting when
the Bloods just not moving as well as it should be or or maybe
maybe the things that damage LDL, there's more LDL to be
damaged, you know, and then that damaged lbl can contribute to
damage as well. It's just like this Cascade of
events in is So like maybe in that state, but if someone's,
you know, living a pretty healthy life that got low levels
of information low levels of oxidative stress and I don't see
why that would be an issue. And, you know, in my book, I
talk about how there's this layer of fourth phase water that
surrounds, the inner layer of the artery.
As long as we're putting ourselves in the right
environment, which is contact with the earth and sun light and
infrared, light and things like that.
And so if that layer is intact, it excludes everything, that's
not it. So it really doesn't matter.
How much LDL is around. It's not getting to the lining
of the artery if that layers attacks.
So it's all about putting yourself in the right
environment. So yeah, so, you know, we'll see
in the future, when some of this, these studies come out,
but I based on anything I look at I don't see it being an
issue. Now.
There is some studies and people with familial
hypercholesterolemia, which is where they haven't genetically
very high LDL levels. And it doesn't seem that they
live any longer or shorter than anybody else.
And one of the studies said that, The people with that
condition who did live shorter, it was likely due to other
lifestyle factors, you know, they were smoking or drinking
and not exercising, or eating processed food diets.
Like it was those things that cause them to live a shorter
life, not their higher LDL. So so, yeah, I don't think that
it would matter, but we'll see. And when other research comes
out, you know, I think so much of it has to be kind of viewed
through the lens of, you know, if someone's following a
ketogenic or lower carb, higher fat, based diet versus more of a
traditional standard. In your mix Diet and then I
would feel like the markers that you're getting are, you know, by
definition going to be highly skewed.
So you can't really, you know, approach the treatment necessary
for the individual through the same lens.
I mean, I'm boggled by people that go and get their their
lipids done and in their their doctor automatically prescribes
them, a Statin without any question whatsoever as to what
their nutrition is, like. My dad just got put on a low
dose that in his, his ldls, not even 200, like it's not
astronomically. Any means, but like, that's not
even part of the general conversation.
When you go to the doctor and they automatically prescribe,
you stands. It seems.
Yeah, and so, it's interesting because if you trace like kind
of the history of what the recommendations are, what LDL
should be or what cholesterol should be.
There was a lot of meetings in the past that were specifically
set up and funded by pharmaceutical companies to
lower the LDL recommendation, you know, to lower what it
should be. And you know, that's more or
less because the Pharmaceutical companies know that if the
recommendations is it for LDL to be under 100, in total question
will be under 200. They can prescribe more statins.
And so you have to take into account but there's that
Financial backing there. But it also tells me that we
really have no idea what LDL is supposed to be what is ideal.
And maybe there is no ideal for any blood marker and that's kind
of the philosophical question. I try and discuss in the last
chapter of my book is that we really don't know because, you
know, if we take, you know, where Menswear for millions of
years which is out of out in nature, eating a natural food
diet, whether it be, you know, more plants or more animals or
whatever. It was, whatever was available
to them. You know, which to me was
probably more animals. What are their blood work look?
Like, you know, like the normal is that we have for our blood
work are based on, you know, healthy populations within the
context of a modern-day society, which is nothing like what for
the majority of time humans. Modern humans have been around.
We're living a Z, right? So so how do we know that?
You know, that those ranges are what are actually normal for
humans or if there even is a normal.
I mean, there was a study that just came out recently maybe
maybe six months ago or so. Then I talk about my book that
that that showed that like it's just an association of study.
So you have to take it with a grain of salt, right?
But it's it showed that the levels of LDL and levels of
total cholesterol that were most.
Released associated with any type of heart disease or
all-cause mortality. In general were a total
cholesterol between 200 and 250, which is above what they say.
It should be in an LDL between 100 and 150, those people who
had those levels. There had the least all-cause
mortality, at least heart disease, cancer least infection
all that kind of stuff. So, so, and that's just an
association so we can't really, you know, say anything from it,
but like it's just interesting because if LDL and total
cholesterol was so causative in heart disease.
You'd think there would at least be an association, you know,
between having higher amounts and and and more disease, right?
But when we don't see that, it's almost like, if these
Association studies not seeing an association is, is way more
useful than seeing one and saying, oh, that's the cosmic
because you can't really prove that, you know, so it's just
interesting to think about that. You know, we humans are trying
to quantify things. That that create disease and
measure those things. So we can assess our risk, but
even like some of the major editors of the major journals,
like Marcia angle, who is the editor of the New England
Journal of Medicine has come out.
And said things like, you know, these, these medical journals
are being used heavily by pharmaceutical companies as
advertising, you know, so it's basically, yeah, it's basically
what can we, what kind of study can we design to?
Show that our product is useful or that, you know, higher levels
of this are problematic so that we can make a drug to bring it
down. You know, like it's they're
being used in that way rather than just purely asking
questions about science and finding the answer, you know,
like it's pretty scary but it just means we have to use that
stuff as part of our, our toolkit, you know, to think how
we should live and how we should be healthy and everything, but
always keep that kind of stuff in mind.
Yeah, would it be? My mind how like you talk to
anybody in the general public and you mentioned the word
cholesterol. They automatically associated
with a negative thing that is is supposed to be lowered, you
know, as much as possible. I think of like cholesterol
being the precursor to testosterone and every cell in
our body having, you know, a layer of fat surrounding like
you don't want to reduce cholesterol to the point where
it's insignificant and I feel like a lot of people are doing
so and their health is not improving as a result.
I mean there, I don't really know.
So to the extent of which, this is true, if it's true at all.
But there has been some studies that show, you know, potentially
adverse effects from too much Statin.
You try it from like a neurological standpoint.
Is there any, is there any weight to that?
Or is that not so much the case? So, definitely.
Yeah, so I go through that and chapter 7, my book, like, you
know, not only all the benefits of cholesterol and what the body
uses it for. But also all the side effects of
statins, you know, there's I just saw a study the other day.
I'm with someone I follow on Instagram.
Newer study. That was showing that, you know,
keeping your LDL too low like, you know, some cardiologists,
like we want it below 70, you know, they aggressively use
statins and then it, basically, the study showed that people who
did that had had cognitive issues because the brain is
largely made up of cholesterol, you know, and so but there's
also, you know, like making making different hormones,
making vitamin D. It's different precursors in the
in the process of making cholesterol are used for
different things like making An internal antioxidants, which are
really important for reducing oxidative stress and
inflammation or, or insulin signal.
Or it's so much chapter 16, you know, one of the precursors is
used for that which, which we want to prevent insulin
resistance, which we talked about already.
So, like, cholesterol is useful for all these different things.
It's necessary for all these different things and if you
lower it to aggressively, you know, you're going to have
issues. And that's why that one
Association of studies. So that that, you know, LDL
between 100 Was associated with the least all-cause, mortality
and disease. No, because your body is using
it for these things. And so, you know, there's
plenty, plenty of evidence that I go through, in my book about
how a statins are not preventing heart to heart attacks or heart
disease. And in some cases, they're even
creating more like atherosclerosis and things.
But also that they're also causing another set of symptoms.
I mean, I'm a I'm a chiropractor.
I do miss them only mainly like neuromusculoskeletal in my
clinic, you know. And I can't tell you how many
people have come in and said, you know, I'm at started having
this pain and I asked him. Okay.
Well what what changed? You know and and lots of them,
not all of them obviously, but lots of them say, well, I
started taking a Statin, you know, and it's causing muscle
pain because cholesterol is essential for for muscle
function. And so if you'll owe her too
much, you're not going to get proper muscle function.
It's going to be painful. So yeah, so all kinds of things,
all kinds of issues and that's just the I got to just highlight
the The I guess the shortsightedness of of the way
pharmaceutical companies and Western medicine, kind of
thinking about things. It's like rather than make the
body work better or figure out. What's why it's not working.
Well, let's try and change one biochemical Pathway to get a
result on a blood work, which is you treating the blood work and
not treating the person. And so, and then yeah, then
you're then you're creating all these other issues because
you're changing one biochemical pathway in a very complex
biological. Logical system that is not
that's not just going to affect one thing.
It's going to affect all kinds of different things in that
complex system. Do you, do you check your blood
pressure regularly? Like do you take that into
consideration through any of this?
No, not really. I mean, I I mean, I've always
had relatively normal or low blood pressure not like
pathologically low. Gadget or anything like that,
but so at that's always been fine, but even, but even then,
like blood pressure is like, we're again, we're very obsessed
with keeping it below. 120 over 80, you know, but, you know,
your blood pressure going up, is it is an indication that your
body's trying to adapt to something to some situation.
You know, that's why there's white coat syndrome when you
have high blood pressure. When you go to the doctor
because you're nervous, right? Like if your blood pressure
didn't go up. When you were nervous, I'd be
concerned, you know, and so we're getting rid of obsessing
over keeping these things where they need to be.
Rather than recognizing that the body needs to be able to attack
the different situations. Now if it tries to adapt to it
and it doesn't come back down. Okay, that's an issue.
You know, like if your blood pressure stays up when you're
when you're just relaxed at home or whatever, under no stress or
whatever. Then okay, maybe that's an issue
or if your blood sugar, you know goes up and never comes back
down or comes back down to slowly.
You know that okay, that's insulin resistance.
That's that's a problem. But people get really obsessed
with these these numbers and what they're supposed to be and
and Instead of measuring the body's ability to adapt which is
taking these static numbers on blood work or Biometrics that
make sense. Yeah total makes total sense
when it comes to you you're going to have probably a little
bit of a different situation because you are type 1 diabetic.
But when it comes to what, you know about the heart, how it
functions always interlinking, you know, equations when it
comes to your nutrition, are you you're pretty much following a
ketogenic low carb approach, right?
Yeah, it's pretty low carb. I so I like, I like to say that
I follow metabolically flexible die, which could be a lot of
different diets, you know, because you could be, you could
eat a vegan, metabolic inflexible diet.
That's possible. You could eat a truly ketogenic
diet were seventy percent of your calories, come from fat and
that could be metabolically flexible.
But the idea is you want to keep your body able to burn fats and
carbohydrates readily. At any time, you know, so if you
and generally that means lower carbohydrate, you know, and not
having tons of carbohydrates all the time, doesn't mean you can
never have them, but you want to keep them to a minimum and you
want to make sure their whole food carbohydrates, but the
majority of your energy comes from fatty acids, and I think
animal fats are way better than plan fast.
And I talked about that in the book, you know, having
cholesterol versus 50 straw, which final straw was like,
basically the plant version of cholesterol, and why Having
cholesterol is way better for you than Phi toaster all.
But, but yeah, so I think that, you know, just eating in a way
that creates metabolic flexibility.
And so, for me, that's, you know, pretty low carbohydrate,
especially trying to control type 1, diabetes.
It's that's really useful for me, just to, to lower the amount
of food that I eat that do raise blood sugar a lot, which are
carbohydrate-rich foods, but I do eat, you know, some berries
and and maybe a sweet potato every now and then think things
like that, but generally it's Pretty low carb, very centered
around animal foods and then, you know, lower carb vegetables
and things like that. That's that's pretty much what I
eat. Yeah.
I mean most people following a
standard American diet or just any the outside of a local cover
approach are never really given their body opportunity to become
fat adapted. So, you know, once you go that
route once you go, you know ketogenic even if only
temporarily your body builds up, those metabolic pathways to A
function and fuel itself with ketones and fatty acids, which
is advantageous. Do you think there's any like
for me? I don't introduce a bunch of
carbs in. I mean, I don't feel better when
I do. I don't perform better when I do
but I don't also think that my body forgets how to metabolize
glucose. So if I was to eat a sweet
potato right now, my body wouldn't know what to do with
it. Like it's not like it forgets
how to do that. But most people never become fat
adapted to begin with apart from when they're born.
So they would, they would benefit from from having these
periods of switching back and forth.
So to speak like you're saying with regard to the metabolic
flexibility. Yeah, it's really interesting
because you can like metabolic metabolic flexible is way worse.
When you become to align on carbohydrates.
It's and if you're eating the standard American diet and
having tons of carbs process carbs, especially all day long,
your body forgets, how to, you know, metabolize fats.
It doesn't forget, but it down regulates those Pathways and it,
it almost forgets how to make ketones, you know, and so when
you start going lower carb, that's why there's like this to
26 weeks, period, where you have this, you know, keto flu kind of
thing, because your body is really having to work hard to
update. Those Pathways again and teach
itself to to, to burn fatty acids, and make ketones for fuel
and stop using carbohydrate now, but you can become metabolic
inflexible. The other way, if you stay in
ketosis for a very long time, but typically, it's like, you
start E2 carbohydrate and, and they've shown this.
Other people are in ketosis for a long time and then they eat a
carbohydrate, their blood sugar does go higher and it looks like
they're in so a distant. But if you did that for like a
day or two, then your body starts burning this carbs again
here and you're just fine. So it's just like the time
period. It takes two to get metabolic
effects. Will from a high carb diet is
way longer and way more damaging, I think because
burning glucose primarily from fuel creates more oxidative
stress and less ATP and things but also it just takes longer to
get there. Whereas, you know, if You were
in ketosis for a long time like your body goes back to burning
carbs pretty quickly. That it's not going to cause any
issues. You know, 100%, 100%.
Yeah, I've had the you know, some people have raised the
question as to if it's damaging to be low carb ketogenic, you
know, except for extended periods of time.
I just I don't see any any indication that that would be
negative. Like there may be that, you
know, 24 or 48-hour period in which you have, you know, hired
blood blood glucose after a postprandial meal with a hiring
carbohydrate, but Like you said that that would typically level
out after just a few days. Yeah, your body would would
readily go to burning those carbohydrates.
Again. Pretty pretty easily.
Yeah, when it comes to, you know, with regard to nutrition,
you were talking about the plant sterols versus, you know, animal
fats. I'd love to kind of dive deeper
net. So, you're more in favor of
saturated animal fats than just loading up on a bunch of
monounsaturated fats or certainly, highly inflammatory
polyunsaturated fats, correct? Definitely.
Yeah, so and it's and it's not necessarily just polyunsaturated
fats, it specifically plant versions of them, you know,
because like when I think about but you know perfect fatty acid
ratio makes a mistake, you know it.
Yeah, it's a just like, you know, grass-fed meat especially,
you know, like that's what we want.
And the problem is that we don't it's not that we don't have
enough Mega threes and we should pop up like Jamaica three pills,
you know, it's it's that we have way too many Omega sixes because
people are eating vegetable oils and things like that.
So that's the problem. So rather than try and even have
to ratio by taking a bunch of Omega-3s or eating a ton of Mega
three, which reads we should really just lowered the omega-6
foods that we're eating. But even even beside besides the
omega-3 versus Omega 6 discussion.
There's cholesterol versus Phi toaster, all which there's, you
know, cholesterol is made for Animals.
It's the fat made for animals and phytosterol is made for
plants as well. Plants use.
It's like the cholesterol version of for plants, right?
And so, We're animals, right? Yeah, so we use cholesterol.
That doesn't mean that our bodies can't use my toaster all
in place of cholesterol that has to, but it would much rather use
cholesterol. And there's actually some
studies that show some evidence that, uh, you know, when we eat
lots of phytosterol, which is like vegetable oils and plant
fats, and things like that, that they compete for absorption in
the gut. And we, absorb less cholesterol,
which is why the, which is why the Like things like margarine
and vegetable oils are touted as Hearts help our heart healthy
because it had it prevents absorption of cholesterol.
Therefore lowering cholesterol in the blood and so they said oh
that's heart healthy, but you know, the fact that lowering
cholesterol is not heart, healthy was never taken into
account. But so it's interesting.
But when your, when your body like, when we're eating tons of
these phytosterols and your body is forced to use, use that in
place of cholesterol. I talk about some studies in my
book that That are that are the looks like it's pretty
problematic. Right?
So one of the one of the issues is that you know, the red blood
cells look like kind of like a donut in their shape.
They they use cholesterol to build up the cell membranes and
that makes them very flexible makes them able to bend around.
So they get into those very small capillaries.
They can kind of squeeze through, right?
And sometimes, they even been themselves a little bit to get
through. However, if our body is forced
to use Phi toaster all to make those Get very rigid.
And so then they try and squeeze through and they end up damaging
things. And so they've done these
studies in rats, not in humans, but they've done these studies
where they feed them more phytosterol and there they
definitely have more Strokes that caused more damage to these
these in arteries, the capillaries because they can't
squeeze through as well. And so I want to build my red
blood cells with cholesterol, not with Fido straw.
And then, there was also this other study where or it was
actually part of the same study, where Are they they did surveys
on people who were going to get a heart valve transplant.
And they asked them how many or how much they basically asked
them like, food surveys or whatever to give an idea of how
much phytosterol, how much plant fat, they were eating versus
animal fat and it showed that the people who ate the most
plant fat when they went in there and they replace the
valve, that the valve, they were going to replace had way higher
amounts of Deposited fight. Oster all around the valve.
And so, and the one who reported eating lab s and was a direct
correlation eating less had much less deposition of phytosterol
around those valves, which is interesting.
Again. It's kind of Association, but
it's a pretty interesting Association.
So it to me, I just think that, you know, where designed to to
eat cholesterol and when it comes to unsaturated fatty
acids, we're supposed to get those from animals as well.
You know, it's not These plant versions aren't serving us as
far as their hiring a mega sixes, which we want to keep
that down a little bit. We still need to make a success
but keep that down to a healthy level and also their
phytosterol, not cholesterol. So basically avoiding all the
soybean oil grape seed, oil grape seed, oil flaxseed oil,
all of these plant-derived fats and oils that are are not really
going to be boding. Well for our the red blood cell,
basically the formation of red blood cells.
Yeah. Yeah.
That's those. That's the big issue because
Like I'm not going to sit here and say, oh don't eat coconut
oil, don't eat olive oil, like pure olive oil.
You gotta make sure it's pure, although not contaminate with
coconut oil, or, with canola oil, or things like that, or
like avocado. I'm not going to say, Don't eat.
Those are definitely healthier things than the standard
American diet, right? The real issue is to the
widespread consumption of the vegetables.
That's what's driving up. The amount of phytosterol were
eating. Its what's driving up, you know,
oxidative damage to those things.
It's what's its that's the real problem.
And so we need to focus. Asan, animal fats.
And if you want to include some plant fats, like coconut oil or
olive oil, things like that. That's fine.
But your diet should have lots of cholesterol and animal fats
in it to make sure that that's the primary thing.
Your body is using to build, not just red blood cells, but also
is really but especially the red blood cells.
Totally. It's been interesting to see
minute. I've been in the space longing
to see how these different types come and go and, you know, for a
while it was like blaming sugar and sugar probably obviously not
optimal but then Lately, it's been this this massive finger
pointing towards highly inflammatory polyunsaturated,
vegetable oils, you know, and they've, they've looked at
graphic graft that out. And so on that, you know, the
Obesity epidemic is kind of more or less correlated to the same
Trend and increased consumption of these oils.
So it's like people are trying to say that's the primary driver
and maybe it is. Maybe it isn't not sure but you
get other people in the space not so much the keto space but
other nutritional spheres that are saying.
Look there is no issue whatsoever with these you know
vegetables eat. Eat them at libitum.
Don't worry about him at all. And I'm just amazed at how much
conflict and controversy there is out there.
Like, how come there's not just some Standalone like we can
agree on this not being optimal, but that doesn't seem to be the
case. Yeah, and I really like, I read
10 Naamans book and I really like his approach because he
breaks it down to, you know, we can sit here and argue about
whether or not these these oils are good or bad and we can
design studies that isolate different.
Balls and try and show that though, the unsaturated fats
aren't bad or whatever. But I like his approach because
it's simple. It's like, you know, when you
eat a food, you know, you wanted to have like, you need, you need
energy, and you need nutrients. And so the energy either food
that has energy and nutrients and that's good.
Right? So, like a steak, it has energy
in the form of fat and, and it has nutrients that sounds of
protein vitamins and minerals. And so that's a good food.
Right, but then you take something like a vegetable oil.
That's just straight energy and that's not good.
That's not, that's never going to satiate your body because it
needs energy and vitamins and minerals.
So I really like that because it breaks it down.
Like that's just logical. It makes complete sense.
Like, and you don't have to go into the Weeds about, you know,
does it? Cause oxidative stress?
Does it cause heart disease? Like what like does do
polyunsaturated fats from plants?
Like do they cause insulin resistance?
Like, which I think they do but, you know, like you don't have to
do all that, you have to say like That's not a natural food.
That's not what humans have been eating for millions of years.
Is this this refined vegetable oil or refined sugar like
anything? Those are just straight energy.
There's no nutrients within which is a problem.
Yeah. No, I like to name is approach
for sure. I've had him on the podcast
several times and we have a lot more common ground that not
people want to inconstancy. He's on one in the Spectrum on
the other but there's a lot more common ground than not.
And I feel like people people in general, just like to operate in
the extremes and people like to do this, incredibly high fat
approach or this incredibly A high-protein approach and that
oftentimes just simply not sustainable and obviously, you
know, whether you're consuming in a surplus or in a significant
deficit is going to have an impact.
I mean if you're consuming at a massive Surplus and that Surplus
is comprised of a lot of Highly inflammatory foods, it's that's
certainly not going to bode well to improving your overall
composition when it's all said and done.
I mean, you got to take all these factors into
consideration. Definitely.
Yeah, and it's, yeah, the extremes kind of people stuck in
it and I understand it too because you know, it's When you,
when you file an extreme thing and you limit everything else,
it it it kind of helps you stay on it, you know, like you can't
say, oh, I'll have that every once in a while because you
getting a end up eating a lot of it, you know, I can understand
for some people that by doing an extreme thing.
It helps them stay more adherent to whatever they're trying to
do. Totally.
Yeah, I get that. But yeah, sometimes I think it's
taken too far and we get dogmatic about it, which is a
problem. And so, so it's better to be,
you know, More open and in your thoughts and things like that.
Yeah, I mean for when it comes to nutrition, I probably just I
try to just make it super simple and tell people.
Hey look, you're going to benefit from having periods of
time in the caloric Surplus. You're going to benefit from
having periods of time in a caloric deficit.
And if each of those times you make that food consists of the
highest quality, you can, you know, from good singling
reading, Wholesome foods with proteins and fats.
You're probably going to be all right, and if I mean that that's
the huge oversimplification, but if people simply just did that,
they would certainly be doing You know, classic 80/20
analysis, and proven things for the general part.
Definitely. Yeah, so what do you got coming,
man? What's in the pipeline for you?
I am obviously speaking of Kita akan.
Nice. And yeah.
Yeah, I'll be there in July, the book comes out.
So my book was originally self-published but I got picked
up by a publisher and so it's being re-released with you know,
I added some things. It's information that I wanted
to add and end. It comes out April. 7th.
It's available for pre-order now, so that's the big push
right now as we're trying to let people know that that's coming
out. And you know the book it talks
about a lot of things that about the from The evolutionary
origins of heart disease and like the evolution of
mitochondria, all the way up to like the diet heart hypothesis
and how that developed with the ancel keys and everything.
But it also talks about, you know, how the heart's, not the
main mover of the blood. And that's not why it's there,
and the mechanisms, by which the blood moves.
It heart talks about why heart cancer is rare and the specific
characteristics of the heart that that make heart cancer,
very rare, and you know what, I think of the true Has of
atherosclerosis, you know, everything from the relationship
of the health of our mouth to heart disease, to to our social
aspect to the autonomic nervous system.
I go into a lot of different aspects of Health, but
definitely hard health. So, that's the big thing for my
nose. At the books, coming out, April
7th. And you know, I'm always I'm
always working on new things. I mean, I'm in the very
beginnings of may be gathering information for the next book.
But but yeah, that's what I got going on.
Now, nice man. Well, I'll be, I'll be getting a
copy. When that comes out for sure,
what is, what is your website resource?
Your health.com resource yelp.com.
Awesome. One more thing.
I meant to ask this earlier, but I totally spaced out the past
two years. We've been hearing a lot about
myocarditis. I know she had a blog post about
that on your site. What is that?
Exactly? And what have you learned about
it over the past two years? Yeah.
So there's there's myocarditis and there's like pericarditis.
So myocarditis is actually inflammation of the heart muscle
itself. Cough.
And it's just like, when this inflammation happens in this
pericarditis, which there's like this kind of like the sac that
surrounds the heart that kind of isolates it, you know, within
within your body and that can become inflamed as well.
And you lot of people talked about that and how it develops.
And you know, there's people have been saying that it's
developed because of the vaccine and stuff, which I wouldn't be
surprised if that was the case, but I don't know if that is the
case or not. But various things can can
relate to this. And it's all we've talked about
inflammation and oxidative stress and over this past hour
and the things that cause that inflammation oxidative stress or
going to cause inflammation anywhere in the body, if you
happen to be susceptible in the area of your heart, like the
heart tissue or or the tissue that surrounds the heart that
the pericardium, then, yeah, you can get inflammation in that
area and the issue with that. Is that is that that like, your
body is put together a very compactly.
And there's no room for things to get inflamed and larger and
things like that. It's Why its my carpal tunnel is
an issue because that nerve gets inflamed and that carpal tunnel
is put together very compactly. And so if something is inflamed,
it starts to cause pain. There's no room for that to
happen. So same kind of thing in the
heart. And so what ended up happening
there is if we get inflammation like that then the heart can't
function as well. You know, it can't contract like
it's supposed to because there's this inflammation pushing on the
side and so that can lead to that can lead to fluid around
the heart, which can cause you because that's that's pushing
into the heart and and Preventing it from Contracting,
which all this stuff will end up like the heart could end up like
kind of reshaping itself a little bit.
So it's instead of the end of it being shaped more like a
football, can be shaped more like a basketball.
And that's what we call it. Like, you know, dilated
cardiomyopathy, where we get a car failure, which basically
means that, you know, the heart is not functioning as a supposed
to, I have my opinions on what the hearts function actually is,
which I outlined in the book and what Ash Drink its blood moving.
But if we're not doing those things, then we get information
of the hearted. It changes the function, the
heart can't Vortex. Like, it's supposed to it.
Can't move blood through itself. Like especially we have to get
pulling up of fluid, which is, you know, a demon like in the in
the arms and legs and things like that.
And and so the but the treatment, you know, is we need
to reduce inflammation. We need to get the blood moving
in other ways, which I talked about like as far as like being
in contact with the Earth and and infrared sauna and things
like that infrared light in general.
But also, we need to remove the things that are causing the
inflammation, which could be environmental toxins could be
stress. Could be poor diet, those types
of things and environmental toxins.
I think are the big ones, which I go through, like, you know
what, those could be. And where to look at in your
life to reduce those exposures in the book.
But yeah, that's that's the inflammation of the heart.
Everybody's been talking about and that's been shown to occur
in certain cases with both the virus and the vaccine.
And it's not like it's targeting the Art specifically it's
causing inflammation everywhere, but it's more pronounced or more
noticeable in the heart because of that compactness.
Yeah. Well, yeah, I mean, and it
really just depends on where someone susceptible to it.
You know, like was there was their heart tissue already
mildly inflamed because they were on a poor diet.
And then that was just the straw that broke the camel's back.
Like I don't know. But you know, any any
inflammatory response to something leaves us, prone to
those types of things. You could have inflammation of
any tissue in the body, really? And and it's just this and some
people like it tends to be the heart and and the heart is a
place where there's a lot of friction, right?
Because the heart is Contracting.
It's moving all the time. And so if you are more prone to
it there and you inflame the body and then you have this
friction as well. That's just going to perpetuate
the inflammation. So the best insurance for
anything. Anytime everyone is to minimize
information whenever and however, possible and improve
metabolic Health, however, possible as well, basically,
Yeah, and, and, and, you know, also keep your blood moving as
well as possible, you know, and so I talk all about that in the
book how how the heart is not necessary this pump.
We think it is. It does some pumping but it it,
you know, it's not the main mover of the blood.
And so there are things we need to be doing that help blood,
keep moving because that's going to take a lot of pressure off
the heart. Take a lot of friction off the
heart. So if they do get into the
inflamed State, the heart's not having to work so hard.
And then therefore creating more pressure and friction.
And so that's things like, I mean, I just did the infrared
sauna this morning because the clear benefits is so many
different benefits for it are from it.
But but that's one of them is that it keeps blood moving very
effectively. So don't get stagnant.
Blood pulling up in areas and more prone to, you know,
clotting or inflammation of things like that.
I know you. Look at HRV quite heavily deep.
Do you put much emphasis on just simply heart rate in general?
Not necessarily. No, I mean again that is
something that I think that, you know, like, we're designed to
adapt if your heart rate doesn't go up in certain situations
that's concerning, you know, in general like heart rate.
If it's if it's high when you're resting that can indicate that
your heart is working too hard for whatever reason and and for
me, that's because it's being forced to act more like a
pumpkin. It's supposed to be.
So it's having to work harder and faster.
So we can do things to take that.
Pressure off of it. But in general, like, you know,
what happens, like measuring your ability to adapt heart rate
to go up and then come back down is way more important than just
what it's just static heartbeat, you know.
Yeah, I think that's a way better measure.
But yeah, heart rate, heart rates important and obviously,
like, highly trained athletes have lower heart rates because
they're the, they become very efficient.
But I also think about what do highly trained athletes.
Do they usually outside more, you know, they're usually doing
Things that that helped take pressure off the heart, they're
not just using their heart more and training it.
They're, they're also using doing things that keep the blood
moving so that the heart doesn't have to work as hard, which is
why it's probably lower. Yeah, so, you know, getting
getting the blood flow and getting some activity and
working out. I mean, all these things like
it's funny like we do all these deep Dives and in nutrition and
you know, heart health and lipid panels and everything and then
like the resounding consensus always seems this be just eat
healthier. Move more and, you know, get out
in the sunshine, get some sleep, like all these super basic
principles that if people just prioritize them their their
ailments would likely be significantly reduced.
Yeah, exactly. And unfortunately many places in
our many people in modern day Society, just those things
aren't prioritized because of the lifestyles that we've
created for ourselves. So it takes some work to
prioritize them for sure. Yeah, totally.
Well, man. I am always learning something
like to talk. When I talk with you.
I'm super excited to be the Look, I will definitely link out
to that your website, your social profiles.
You are a wealth of knowledge on so many different levels, man.
That's always pushing each other podcast.
If there's anything I can do for you and just let me know.
Hey, man, thanks for having me. Take care, brother.